Download Multiple Sklerose: Krankheitsbewaltigung - by Sabine Twork, Joachim Kugler PDF

By Sabine Twork, Joachim Kugler

Das vorliegende Buch befasst sich mit der Krankheitsbew?ltigung bei Multipler Sklerose und versucht verschiedene Aspekte der Krankheitsbew?ltigung abzubilden. Die Sichtweisen aus Klinik, Niederlassung sowie Grundlagenforschung erg?nzt es um die Perspektive der Versorgungsforschung. Die Autoren m?chten zu einem gr??eren Verst?ndnis sowie einer besseren Unterst?tzung MS-Betroffener bei ihrer Krankheitsbew?ltigung beitragen.

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Additional info for Multiple Sklerose: Krankheitsbewaltigung - Therapiemotivation - Lebensqualitat: Erste Ergebnisse zum Coping-Training MS-COPE

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Eur J Immunol 29: 2705–2714 55. Ritter AT et al. (2005) Sicherheit und Verträglichkeit von anti 111 In-CD-20-Fab zum Nachweis von B-Zellen in aktivierten Gehirnläsionen bei MS. Akt Neurologie 32: 1055 56. Niehaus A, Shi J, Grzenkowski M, Diers-Fenger M, Hartung HP, Toyka KV, Bruck W, Trotter J (2000) Patients with active relapsing-remitting multiple sclerosis synthesize antibodies recognizing oligodendrocyte progenitor cell surface protein: implications for remyelination. Ann Neurol 48: 362–371 57.

Results of adrenal and pituitary stimulation tests in patients with multiple sclerosis. Acta Neurol Scand 1972; 48: 467–478 13. Maida E, Summer K. Serum cortisol levels of multiple sclerosis patients during ACTH treatment. J Neurol 1979; 220: 143–148 14. Teasdale GM, Smith PA, Wilkinson R, Latner AL, Miller H. Endocrine activity in multiple sclerosis. Lancet 1967; 1: 64–68 15. Millac P, Cook DB, Chase K. Endocrine function in multiple sclerosis. J Neurol Neurosurg Psychiat 1969; 32: 414–418 16.

Dies legt den Schluss nahe, dass zunehmende Schädigung des ZNS zu einer Aktivierung oder Enthemmung des HHN-Systems führt. Als Mechanismus dafür kann die Schädigung von Fasern aus anderen Hirnregionen diskutiert werden, die hemmenden Einfluss auf den Hypothalamus haben; solche Einflüsse sind allerdings bisher hypothetisch geblieben [32]. Die Alterna- 36 2 Kapitel 2 ∙ Hormonelle Aspekte der Stressreaktion tive, eine Koaktivierung der HHN-Achse durch CRH und Vasopressin ist wahrscheinlicher und experimentell gut belegt [33].

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