Download Computer assisted optimization of cardiac resynchronization by Raz Miri PDF

By Raz Miri

The efficacy of cardiac resynchronization treatment (CRT) via biventricular pacing (BVP) has been proven by way of quite a few reviews in sufferers being affected by congestive center failure. so one can in achieving a suggestion for optimum remedy with BVP units, an automatic non-invasive approach in accordance with an electrophysiological laptop version of the center is gifted. The offered examine investigates an off-line optimization set of rules in accordance with various electrode positioning and timing delays

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In general, atrial flutter should be treated the same as atrial fibrillation. Atrial flutter is considerably more sensitive to electrical cardioversion than atrial fibrillation, and usually requires a lower energy shock. Because of the reentrant nature of atrial flutter, it is often possible to ablate the circuit that causes atrial flutter. 3. Ventricular fibrillation (VF): Very serious rhythmic abnormality in ventricular myocardium reveals uncoordinated, chaotic contractions. Multiple impulses travel erratically in all directions within the ventricles.

This may be attributable to increased wall stress due to late activated myocardial segments but the underlying mechanisms require further investigation. Outcomes in major randomized CRT trials including MIRACLE, MIRACLE-ICD, COMPANION, CARE-HF show an improvement in NYHA functional class, ejection fraction and quality of life [150]. 44 Chapter 3. Cardiac Resynchronization Therapy In most studies, echocardiographic parameters including LVEF, left ventricular end diastolic diameter (LVEDd) and regional wall motion were recorded before CRT implantation (baseline) and after implantation weeks or months later (follow up).

The presence of mitral regurgitation results in an increasing volume overload on the overburdened left ventricle that further contributes to remodeling, the progression of disease, and to symptoms. 4. 27. Ventricular remodeling in diastolic and systolic heart failure [133]. focus of therapy. 4. Hypertensive heart disease: caused by high blood pressure. Hypertensive heart disease is a late complication of hypertension (high blood pressure) in which the heart is affected. High blood pressure increases the heart’s workload and over time, this can cause the heart muscle to thicken.

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