Download Cellular Therapy for Neurological Injury by Jr., Charles S. Cox PDF

By Jr., Charles S. Cox

Cellular treatment for Neurological Injury discusses the present prestige of mobile remedy for neurological problems. the first parts of concentration comprise tense mind damage, stroke (ischemic and hemorrhagic), and spinal wire harm. The publication explores mobilephone treatment methods to those and different stipulations, whereas discussing present advances and a literature assessment within the context of a box that's relocating swiftly.

The e-book offers a translational concentration, addressing obstacles and possibilities to relocating ahead. The mechanisms of harm are explored, in addition to how those mechanisms impression the kind of cellphone remedy used, the course of supply, and dosing routine. Written through leaders within the box, this is often a useful source for clinicians and researchers alike.

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Surgery 144 (5):803–813. , 2009. Intravenous mesenchymal stem cell therapy for traumatic brain injury: Laboratory investigation. J Neurosurg. 110:1189. , 2014. Sox2-mediated conversion of NG2 glia into induced neurons in the injured adult cerebral cortex. Stem Cell Reports. 3:1000–1014. , Marino, D. , 2005. Experimental autoimmune encephalomyelitis repressed by heral and central nervous systems. Results Probl Cell Differ. 48:339–351. , Murray, G. et al. 2006. com). Acta Neurochir Suppl. 96:17–20.

2007. Modulation of the cAMP signaling pathway after traumatic brain injury. Exp Neurol. 208 (1):145–158. , 2012. Mortality and long-term functional outcome associated with intracranial pressure after traumatic brain injury. Intensive Care Med. 38 (11):1800–1809. , 2006. Persistent and high levels of Hes1 expression regulate boundary formation in the developing central nervous system. Development (Cambridge, England). 133:2467–2476. , Gordon, M. et al. 2014. Intra-arterial immunoselected CD34+ stem cells for acute ischemic stroke.

In a series of 97 patients, Tian and associates were able to demonstrate that they could safely harvest and administer autologous MSC using as much as 5 mL (1 × 106 cells/mL) via lumbar puncture in patients with persistent vegetative state or other deficits 1 month after injury. In this series of patients, the greatest improvements were seen in younger patients who underwent treatment closer to the time of injury (Tian et al. 2013). The use of allogenic, umbilical cord MSC was evaluated in patients with chronic deficits following TBI.

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